Energy Efficiency

Metabolic Health for Dairy Ruminants

Transition Period, Negative Energy Balance (NEB) & Energy Efficiency

In the realm of livestock farming, understanding and managing the energy balance in ruminants, especially during critical periods like pre-calving, is fundamental to maintaining animal health and farm productivity. The pre-calving period is marked by a complex interplay between the animal’s increasing energy needs and its actual energy intake. As ruminants approach calving, their energy expenditure escalates significantly to support fetal growth and lactation preparation. However, this heightened demand often coincides with a decrease in Dry Matter Intake (DMI), creating a challenging mismatch.

This energy deficit compels the ruminant to mobilise its fat reserves as an alternative energy source rapidly. While this is a natural adaptive response, excessive and rapid fat mobilisation can lead to a detrimental spiral. In an attempt to compensate for the energy shortfall, the animal’s body breaks down fat reserves too quickly and in large amounts. This rapid fat breakdown results in an accumulation of fatty acids in the liver and an overproduction of ketone bodies, leading to metabolic disorders such as fatty liver syndrome and ketosis.

Such disorders not only impact the health and welfare of the animals but also have far-reaching consequences on their productivity. The spiralling effect of this energy imbalance can lead to a cascade of health issues, reduced milk yield, and reproductive challenges post-calving. Recognising and addressing this energy mismatch before calving is crucial. It involves careful nutritional management, monitoring of body condition, and strategic feeding practices to ensure a smoother transition for the ruminant into the lactation period, thereby safeguarding both the animal’s health and the farm’s productivity.

Reproductive Health and Risks Post-Energy Imbalance

Energy imbalances experienced by ruminants during the crucial transition period have a profound impact on their reproductive health. This period, which encompasses the time shortly before and after calving, is critical for setting the stage for the cow’s future reproductive and lactation performance. Overconditioned cows, in particular, are at a heightened risk of experiencing reproductive complications in subsequent cycles. These complications often manifest as extended calving intervals, frequent need for repeated breedings, and notably lower pregnancy rates. Such issues are primarily a consequence of the metabolic stress encountered during the pre-calving phase.

These reproductive challenges have far-reaching implications, including a decrease in milk production and an increase in culling rates. Poor fertility and suboptimal performance are significant factors that drive these increased culling rates, leading to substantial economic burdens on farm operations. The reduced milk yield not only affects the immediate lactation cycle but can also have lingering effects on the herd’s overall productivity.

To address these challenges, effective management strategies during the transition period are vital. These include regular and thorough health monitoring practices, encompassing body condition scoring and detailed reproductive assessments. Monitoring body condition helps in identifying cows that are at risk of becoming overconditioned and allows for timely dietary adjustments. Similarly, reproductive assessments enable early detection of potential fertility issues, allowing for prompt intervention.

In addition to these measures, implementing a comprehensive nutritional plan tailored to the specific needs of each cow during this period is crucial. This involves ensuring adequate energy intake to meet the increased demands of late gestation and early lactation, while avoiding excessive energy that leads to overconditioning.

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Pregnancy Toxaemia in Small Ruminants

In small ruminants, the threat of Pregnancy Toxaemia during late gestation poses significant risks. This metabolic disorder, precipitated by a Negative Energy Balance (NEB), results from increased energy demands and reduced appetite, leading to a lower Dry Matter Intake (DMI). Excessive fat mobilization, particularly in over conditioned animals, causes a buildup of Non-Esterified Fatty Acids (NEFA) in the liver, triggering an overproduction of ketone bodies. This exacerbates the issue by further depressing the appetite and causing a dramatic plunge in DMI.

The consequences of Pregnancy Toxaemia are profound and multi-faceted. Miscarriages or the death of the ewe before lambing are common, leading to total loss of offspring and potential loss of the mother. Surviving ewes often suffer from reduced or impaired milk yields and shorter lactation periods. Their impaired condition also leads to underperformance in subsequent cycles. Overfeeding during recovery can result in the ewe becoming overweight, increasing the risk of recurrence in the next gestation period. For grass-fed sheep, the weakened state post-toxaemia delays their return to the breeding cycle, making them outliers in the herd and disrupting the breeding schedule.

Energy Needs Rise

Energy needs increase before and right after calving

Overconditioning

Extended calving intervals at lower production lead to cow’s overconditioning

Anovulation

Cows well beyond the VWP won’t cycle (no sign of estruos)
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Fat Mobilization

Fat mobilization to reduce NEB leads to metabolic disorders (fatty liver, ketosis)

Depressed DMI

Metabolic disorders further decrease DMI aggravating NEB

Hormonal Axis

Severe NEB disrupts the reproductive hormonal axis

Energy Needs Rise

Energy Requirements keep building up

DMI Plunges

Excess ketone bodies depress appetite further and lead to DMI plunge

Fatty Liver

Liver fills with NEFA* leading to excess ketone bodies production
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Depressed DMI

Reduced appetite and lower DMI reduce energy input

NEB Kicks In

Negative Energy Balance (NEB) sets in

Fat Mobilization

Excessive fat mobilization (especially when overconditioned)

Energy Needs Rise

Energy Requirements keep building up

DMI Plunges

Excess ketone bodies depress appetite further and lead to DMI plunge

Fatty Liver

Liver fills with NEFA* leading to excess ketone bodies production
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Depressed DMI

Reduced appetite and lower DMI reduce energy input

NEB Kicks In

Negative Energy Balance (NEB) sets in

Fat Mobilization

Excessive fat mobilization (especially when overconditioned)

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